Interpreting the Mechanism of APOE (p.Leu167del) Mutation in the Incidence of Familial Hypercholesterolemia; An In-silico Approach

Fig. (1) (A). Protein 3D model of wild type APOE with emphasis on the Leu167aa position (B). Protein 3D model of the APOE p.Leu167del with emphasis on Leu167aa deletion from the model (C). Protein 3D model of the LDLR.